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The ic-STZ-injected rats did not have elevated blood glucose or insulin levels, and pancreatic architecture and insulin immunoreactivity were similar to control, yet their brains were atrophied and had striking evidence of neurodegeneration with cell loss, gliosis, and increased immunoreactivity for p53, activated GSK-3β, phospho-tau, ubiquitin, and APP-Aβ.

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Excess secretion of these hormones may contribute to insulin resistance in particular settings, but does not account for the vast majority of insulin resistant states.

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The use of glucose and fructose calculated from sweetener disappearance increased in parallel during this time period ( Figure 2).

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The conditional muscle insulin receptor knockout mouse has impaired insulin signaling and decreased insulin-dependent glucose transport, without systemic insulin resistance, but develops metabolic syndrome and dyslipidemia (5).

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Grb10 interacts differentially with the insulin receptor, insulin-like growth factor I receptor, and epidermal growth factor receptor via the Grb10 Src homology 2 (SH2) domain and a second novel domain located between the pleckstrin homology and SH2 domains".

Use: To improve glycemic control in adult patients with diabetes mellitus.

Insulin resistance is an abnormal condition whereby the receptors of the cells that respond to insulin have lost sensitivity and become "resistant" to insulin.

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Tsui E, Barnie A, Ross S, Parkes R, Zinman B: Intensive insulin therapy with insulin lispro: a randomized trial of continuous subcutaneous insulin infusion versus multiple insulin injections.

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In the early stages of insulin resistance, the pancreas compensates by increasing the secretion of insulin into the bloodstream in an attempt to overcome defects in peripheral insulin action.

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These analogs function very well in continuous subcutaneous insulin infusion (CSII) systems (insulin pumps), resulting in lower HbA 1c concentrations and postprandial blood glucose levels than regular insulin, with less hypoglycemia.

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Metabolic syndrome (MS) is a collection of cardiometabolic risk factors that includes obesity, insulin resistance, hypertension, and dyslipidemia.

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The postexercise oxygen uptake in excess of that required at rest has been termed excess postexercise oxygen consumption (EPOC).

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This interest has yielded insulin analogues that are characterized by action profiles that afford more flexible treatment regimens with a lower risk of the development of hypoglycemia ( Table 1 Duration of Action of Standard Insulins and Insulin Analogues.

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Massachusetts General Hospital and colleagues found that among 101 HIV-positive people in the Framingham Offspring Study (a large study of cardiovascular risk), those with body fat changes were more likely to have impaired glucose tolerance (evidenced by elevated insulin levels and increased glucose levels after a glucose tolerance test) and frank diabetes.

Again, using a similar 2-wk paradigm, SIT increased glucose infusion rate by 21% in young men and women 72 h after the last training session during EHC ().

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Regular, also known as short-acting insulin, takes about 30 minutes to reach the bloodstream.

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